Re: Auditory efferents and phase locking (=?ISO-8859-1?Q?Jos=E9_Ignacio_Alc=E1ntara?= )


Subject: Re: Auditory efferents and phase locking
From:    =?ISO-8859-1?Q?Jos=E9_Ignacio_Alc=E1ntara?=  <jia10@xxxxxxxx>
Date:    Thu, 1 Sep 2011 04:26:34 +0100
List-Archive:<http://lists.mcgill.ca/scripts/wa.exe?LIST=AUDITORY>

On Sep 1 2011, Matt Flax wrote: If you accept that some or many of the ageing/senescence effects are due to mitochondrial dysfunction*, then you might expect those (sensory) neurones with the highest energy demands to be particularly vulnerable: presbyacusis might simply be the result of OHC/IHC/AN damage/apoptosis? *e.g., Oxidative damage during aging targets mitochondrial aconitase LIANG-JUN YAN, RODNEY L. LEVINE, AND RAJINDAR S. SOHAL Proc. Natl. Acad. Sci. USA Vol. 94, pp. 11168-11172, October 1997 Jose >p.s. If it was to protect, then why is Presbycusis so bad ? >On Wed, 2011-08-31 at 16:54 -0700, Richard F. Lyon wrote: >> Perhaps the >> gain-control role is primarily protective, as many have speculated? > -- José _________________________________ José Ignacio Alcántara, PhD University Lecturer Department of Experimental Psychology University of Cambridge Downing Street Cambridge CB2 3EB United Kingdom Tel: +44 (0)1223 764412 Fellow of Fitzwilliam College Storeys Way Cambridge CB3 0DG United Kingdom Tel: +44 (0)1223 472126


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